The Clinical Pattern
In my recent QCS scans, I have noticed a puzzling pattern of hypervitaminosis D, increased calcium, parathyroid hormone and associated influences. This leads me to wonder — do we need to re-examine the role of Vitamin D, especially with direct or indirect exposure to the COVID spike protein?
These cases fit into two predominant categories. Those with A+ blood type had hypervitaminosis D and typically hypercalcemia. O+ blood type clients typically presented with just hypercalcemia. In my observations, 99% of the time clients fit into these two groups.
Mast Cells & COVID Hyperinflammation
In the early days of COVID, much discussion centred on Vitamin D deficiency creating increased morbidity and mortality. The ACE-2 receptor was thought to be influenced in part by Vitamin D cell and nuclear receptors on macrophages — leading many to mega-dose on Vitamin D. But that isn’t the whole story.
In November 2020, a group of doctors and mast cell researchers noted: “Much of Covid-19’s hyperinflammation is related to inflammation which mast cell [MC] activation can drive. Drugs with activity against MCs or their mediators have preliminarily been observed to be helpful in Covid-19 patients.” PMID: 32920235
QCS scan data illustrating the Vitamin D and calcium patterns observed across client populations.
Mast cells discharge an inflammatory soup of histamine, leukotrienes, prostaglandins and cytokines when triggered. Present in most tissues — including the lung, gut and brain (as microglial cells) — a dysregulated degranulation of mast cells results in allergy or hyper-inflammation.
Spike Protein & Mast Cell Degranulation
In December 2021, Wu et al. published “SARS-CoV-2-triggered mast cell rapid degranulation induces alveolar epithelial inflammation and lung injury.” PMID: 34921131
The spike protein binds to the ACE2 receptor on mast cells, triggering degranulation — relevant to both COVID infection and mRNA vaccine exposure.
The VDR Hypothesis — Why Vitamin D Looks Elevated
My belief is that the Vitamin D Receptor (VDR) function may be overwhelmed by dysregulated spike protein responses, causing increased serum Vitamin D levels — and in QCS scanning, causing calcium build-up in the body, also known as granulomatous disease. PMID: 33864942
In more than half of these cases, clients were NOT consuming Vitamin D despite elevated serum levels. Analysis indicated the client did need Vitamin D — however could not absorb it. This is a fundamental departure from conventional understanding of Vitamin D deficiency in the context of COVID infection.
A 2017 study found that mast cells activate spontaneously in a Vitamin D-deficient environment, and Vitamin D levels are inversely correlated with COVID severity. PMID: 27998003 Maintaining adequate plasma Vitamin D depends not just on diet and sun exposure, but on reduction in pollutant, pesticide and glyphosate exposure and a healthy microbiome.
The Gut–Mast Cell–Vitamin D Triangle
Inflammatory endotoxin LPS, produced by certain gut bacteria, drives chronic inflammation via endotoxemia. Chronic inflammation depletes Vitamin D. LPS upregulates Vitamin D-metabolising enzymes (CYP27B1 and CYP24A1) in white blood cells, accelerating Vitamin D degradation.
Mast cell stabilisation requires maintaining a healthy gut barrier to prevent LPS leakage into the bloodstream. Is it possible that one answer to hypervitaminosis D lies in leaky gut — made worse by dysregulated mast cells that themselves contribute to gut barrier dysfunction, potentially fuelling their own destabilisation?
Additional mast cell destabilisers include endocrine-disrupting herbicides, environmental toxicities, power-frequency EMFs and psychological stress via stress hormones and neurochemicals.
Clinical Observations by Blood Type
Distinct patterns observed in QCS scanning: hypervitaminosis D predominantly in A+ clients, hypercalcemia in both A+ and O+ populations.
Exposure to the spike protein appears to cause dysregulation of both Vitamin D and calcium in A+ clients. In O+ clients, Vitamin D dysregulation was not apparent — however, hypercalcemia was consistently present. In both cases, hypercalcemia symptoms predominated: nausea, GI issues, fatigue despite inability to sleep, restless legs, achy joints, vision issues, cloudy thinking and neurological changes.
Clinical Approach & Alive Innovations Support
Healing from long-COVID, COVID vaccine injury, or preparing to handle COVID exposure uneventfully should include mast cell care and microbiome support to regulate Vitamin D absorption and calcium metabolism.
In my lifetime I have rarely seen Vitamin D toxicity — especially in those not supplementing. Seeing clients in their 30s–50s with calcium issues where thyroid and parathyroid function is clinically normal is extraordinary. The spike protein appears pervasive in these observations, clearly linked with these conditions — creating a Trilogy of Trouble.